Genetic Distance |
Surname |
Kit# |
Most
Down-
stream
Positive
SNP
Actually
Tested |
|
Panel 1 — Standard Markers
1-12 |
|
Panel 2 — Standard Markers
13-25 |
|
Panel 3 — Standard Markers
26-37 |
|
Panel 4 — Standard Markers
38-67 |
|
Panel 5 — Standard Markers
68-111 |
|
Known
Lineage |
Temporary and Arbitrary Modification (8 Dec 2018):
to demonstrate the reason we need everyone to upgrade to 111 markers and
produce a better family modal value, I've made some arbitrary changes to
the Family Model (FM) in Panel 5 in the table above. In fact,
these changes are reasonable ones; and one or more of them may, in the
end, turn out to be a true FM value.
Because #345432 was the only STROPE/STROUP to have tested 111 markers,
I had previously used his values verbatim in Panel 5 as the FM giving
him a GD of zero from the FM. Having a sample size of just one (n=1)
makes calling this a "modal" value somewhat absurd. Doing this caused
the MILLER to appear to have a GD of 6 from the FM in Panel 5, giving him
a Total GD of 8, thus making him only a "borderline" match with the family.
However, if we assume they do have a near common ancestor, it is reasonable
to assume that #345432 and the MILLER are equidistant from him in terms
of generations, and thus would have acquired a roughly equal number of
mutations in that descent — and, normally, this will be the actual case
for descendants of the same near common ancestor.
Until the other three members of this family upgrade to 111 markers,
we cannot know what the actually FM is — especially as a sample size of
four or five is still not great. So I have, in the table above, arbitrarily
changed some FM values to split the GD in Panel 5 as evenly as possible
between #345432 and the MILLER. Having done so, the MILLER becomes
a solid member of this family. PLEASE, everyone upgrade to
111 markers. This is exactly the kind of genealogical problem that
DNA testing can resolve. |
Our subjects have a rare haplotype, as evidenced by the GD (Genetic
Distance) of their family modal haplotype from the I2-P37 modal haplotype
(42!). They have only one match with anyone else at any level, a
MILLER. With a 65/67 match to the family modal haplotype, it was
looking like the MILLER had an NPE
and was really a STRAUB. However, at 111 markers that match fell
away and became "borderline." There is little doubt they have a near
common ancestor, especially given the rarity of their haplotypes, but the
question remains whether this is an NPE or simply a connection before the
period of surname adoption, in Germany. In other words, is he actually
a German MÜLLER, rather than an English MILLER?
What's needed, now, is for the three individuals who've only tested
67 markers to upgrade to 111 markers — only one individual tested to 111
markers does not make for a strong modal. The best way to do this
is to take the BigY-500, which includes the upgrade to 111 markers; the
MILLER also needs to take the BigY. SNP results from the BigY would
tell us whether the STR match represents relationship or coincidence.
FTDNA's annual Holiday Sale is in effect until December 31st, so this
would be a good time to take the tests. The sale price for an upgrade
from 67 markers to 111 markers is $89 (reg. $129); an upgrade from 67 markers
to the BigY, including the upgrade to 111 markers, is $399 (reg. $499);
the upgrade from 111 markers to the BigY is $349 (reg. $449).
I know the BigY is expensive, but in the end it's cheaper than testing
SNP packs and individual SNPs, which only test known SNPs. The BigY
is an exploratory test and will find new SNPs unique to your family and
to you, individually. This is the level of testing we need to answer
questions at the family/individual level. |
This family has an interesting history, which has been compiled by
descendant, Bonita El ine STROPE, in her book,The
Strope Family from Germany to America. |
#345432 received a gratis backbone SNP test (P37.2+) from FTDNA to
determine the haplogroup for this rare haplotype. |
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• The descendants of Clarence Charles share a mutation from 12
to 11 at DYS442 (magenta table cells), which had to have occurred no later
that Clarence's son and possibly much earlier. Testing of further
cousins could pinpoint the generation in which the mutation actually appeared,
making this mutation a branch identifier for the family.
• The paper documentation connecting Benjamin4 and
William II3 is weak; research continues. |
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